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The biology of human longevity : inflammation, nutrition, and aging in the evolution of life spans / Caleb E. Finch.

By: Material type: TextTextPublisher: Amsterdam ; Boston : Academic Press, c2007Description: xiii, 626 pISBN:
  • 9780123736574 (hard cover : alk. paper)
  • 0123736579 (hard cover : alk. paper)
Subject(s): DDC classification:
  • 612.6/8 22
LOC classification:
  • QP85 .F466 2007
Contents:
Ch. 1. Inflammation and oxidation in aging and chronic diseases -- Ch. 2. Infections, inflammogens, and drugs -- Ch. 3. Energy balance, inflammation, and aging -- Ch. 4. Nutrition and infection in the developmental influences on aging -- Ch. 5. Genetics -- Ch. 6. The human life span : present, past, and future.
Review: "The Biology of Human Longevity: Inflammation, Nutrition, and Aging in the Evolution of Lifespans synthesizes several decades of top research, and expands a number of existing major theories, including the Barker theory of fetal origins of adult disease to consider the role of inflammation and Harmon's free radical theory of aging to include inflammatory damage. Future increases in lifespan are challenged by the obesity epidemic and spreading global infections that may reverse the gains made in lowering inflammatory exposure."--BOOK JACKET.
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Includes bibliographical references and index.

Ch. 1. Inflammation and oxidation in aging and chronic diseases -- Ch. 2. Infections, inflammogens, and drugs -- Ch. 3. Energy balance, inflammation, and aging -- Ch. 4. Nutrition and infection in the developmental influences on aging -- Ch. 5. Genetics -- Ch. 6. The human life span : present, past, and future.

"The Biology of Human Longevity: Inflammation, Nutrition, and Aging in the Evolution of Lifespans synthesizes several decades of top research, and expands a number of existing major theories, including the Barker theory of fetal origins of adult disease to consider the role of inflammation and Harmon's free radical theory of aging to include inflammatory damage. Future increases in lifespan are challenged by the obesity epidemic and spreading global infections that may reverse the gains made in lowering inflammatory exposure."--BOOK JACKET.

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